2. Membrane Transporter/Ion Channel
  3. ATP Synthase
  4. BMS-199264 hydrochloride

BMS-199264 hydrochloride 是 F1F0 ATP 水解酶抑制剂 (IC50=0.5 μM)，对 F1F0 ATP 合酶没有抑制作用。BMS-199264 hydrochloride 在缺血期间选择性地抑制 ATP 下降以减少心脏坏死。BMS-199264 hydrochloride 还可以增强再灌注后收缩功能的恢复。

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BMS-199264 hydrochloride Chemical Structure

BMS-199264 hydrochloride Chemical Structure

CAS No. : 186180-83-6

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BMS-199264 hydrochloride 相关抗体

Top Publications Citing Use of Products

  • 生物活性

  • 纯度 & 产品资料

  • 参考文献

生物活性

BMS-199264 hydrochloride is an inhibitor of F1F0 ATP hydrolase (IC50=0.5 μM) without inhibitory effect on F1F0 ATP synthase. BMS-199264 hydrochloride selectively inhibits ATP decline during ischemia to reduces cardiac necrosis. BMS-199264 hydrochloride also enhances the recovery of contractile function following reperfusion[1].

体外研究
(In Vitro)

BMS-199264 hydrochloride (1 μM、3 μM、10 μM) 在 25 分钟全脑缺血后 30 分钟再灌注后以浓度依赖性方式增加挛缩发作时间并减少 LDH 释放[1]。
BMS-199264 hydrochloride (3 μM) 对 ATP 合成酶或水解酶活性有不同的影响，分别为 0.23 μMATP/min/mg 和 0.18 μMATP/min/mg[1]。

MCE has not independently confirmed the accuracy of these methods. They are for reference only.

BMS-199264 hydrochloride 相关抗体:
分子量

567.53

Formula

C26H32Cl2N4O4S
CAS 号
运输条件

Room temperature in continental US; may vary elsewhere.
储存方式

Please store the product under the recommended conditions in the Certificate of Analysis.
纯度 & 产品资料
参考文献

BMS-199264 hydrochloride 相关分类

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Keywords:

BMS-199264186180-83-6BMS199264BMS 199264ATP Synthasecardiac necrosisischemiareperfusionF1F0 ATP hydrolaseInhibitorinhibitorinhibit

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